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. 2015 Jul;22(7):336–343. doi: 10.1101/lm.038083.115

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© 2015 Venna et al.; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first 12 months after the full-issue publication date (see http://learnmem.cshlp.org/site/misc/terms.xhtml). After 12 months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 5. — GSK-3β inhibition along with TAK1 inhibition has no additional neuroprotective effects compared with GSK-3β inhibition alone. (A) Quantification of infarct volume reveals no significant additional neuroprotective effects of GSK-3β inhibition in combination with TAK1 inhibition; (n = 9 per group). (B) Combined inhibitor treatment did not improve the percentage of time spent with a novel object compared with GSK-3β inhibition alone. (*) P < 0.05; (n = 9 per group). (C) Combined inhibitor treatment did not improve the amount of time spent on rotarod compared with GSK-3β inhibition alone suggesting a lack of additional benefit on post-stroke learning recovery. (*) P < 0.05.