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. 2015 Jun 24;6:436. doi: 10.3389/fpls.2015.00436

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Copyright © 2015 Wang, Mao, Yang and He.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Secondary cell wall thickening of anther endothecium in the plants with TCP24 silencing. (A) Relative expression of miR319a-targeted TCP genes in flower buds of jaw-D mutants. (B,C) Transverse sections of the anthers in the wild type (B) and jaw-D (C) plants. (D–F) Anthers of the wild type (D) and the p35S:TCP24SRDX (E,F) plants. Arrows indicate protuberances. (G–I) Phloroglucinol staining of anthers showing secondary cell wall thickening of anther endothecium in the wild type (G) and the p35S:TCP24SRDX (H,I) plants (the strong phenotype in the left and weak phenotype in the right). Scale bars: 10 μm in (B,C).