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. 2015 Apr 14;43(9):4408–4428. doi: 10.1093/nar/gkv281

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© The Author(s) 2015. Published by Oxford University Press on behalf of Nucleic Acids Research.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 2. — (A) Percentage of reads mapped to coding and non-coding genes in myoblasts and myotubes treated with harringtoning (Har) or cycloheximide (Chx). (B) Read-length distribution of footprints mapping to protein-coding genes (top), non-coding genes (including small and long non-coding genes) or only to lincRNAs (bottom). (C) Read-length distribution of footprints mapping to Malat1, Snhg1, Rnu11 and H19. (D–G) Coverage patterns for Malat1, Snhg1, Rnu11 and H19 in harringtonin (top traces) and cycloheximide (bottom traces) treated myoblasts.