Skip to main content
. 2015 Mar 21;6(13):11547–11560. doi: 10.18632/oncotarget.3413

Figure 3. Restoration of BCL6B expression inhibited proliferation, induced aopotosis and G1/S arrest in HCC cells.

Figure 3

A. Colony formation in BCL6B re-expressed and unexpressed HepG2 and SNU449 cells. Each experiment was repeated for three times. **p < 0.01. B. Growth curves of BCL6B re-expressed and unexpressed HepG2 and SNU449 cells analyzed by CCK-8 assay. Each experiment was repeated for three times. **p < 0.01. C. Representative apoptosis data analyzed by flow cytometry in BCL6B re-expressed and unexpressed HepG2 and SNU449 cells. Each experiment was repeated for three times. **p < 0.01. D. Cell cycle distribution in BCL6B re-expressed and unexpressed HepG2 and SNU449 cells. Each experiment was repeated for three times.