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. 2015 Jun 2;16(6):12424–12435. doi: 10.3390/ijms160612424

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© 2015 by the authors; licensee MDPI, Basel, Switzerland.

This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution license (http://creativecommons.org/licenses/by/4.0/).

PMC Copyright notice

The focused panel gene expression profile of Ishikawa cells was studied using the RT² Profilter^TM PCR array following four-hour exposure to vehicle (DMSO) or PCAE 2 mg/mL. Fold changes greater than two (up-regulation) or smaller than 0.5 (down-regulation) were considered substantial. Fold changes were calculated by dividing expression levels in PCAE-treated cells by expression levels in the vehicle-treated cells. BAG3: BCL-associated athanogene 3; CASP4: Caspase 4, apoptosis-related cysteine peptidase; CASP5: Caspase 5, apoptosis-related cysteine peptidase; HRK: Harakiri, BCL2 interacting protein; BCL2A1: BCL2-related protein A1; CASP1: Caspase 1, apoptosis-related cysteine peptidase; CASP14: Caspase 14, apoptosis-related cysteine peptidase, NOL3: Nuclear protein 3; PYCARD: PYD and CARD domain containing; TNF: Tumor necrosis factor, TNFRSF10A: Tumor necrosis factor receptor superfamily, member 10a; CD27: CD27 molecule, TNFSF10: Tumor necrosis factor receptor superfamily, member 10; TP73: Tumor protein p73.