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. 2015 Jul 3;10:87. doi: 10.1186/s13000-015-0324-y

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© Abedinzadeh et al. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Micrographs showing the coronary tree from patients who died of acute myocardial infarction (AMI) . Panel (a) demonstrates the fatty deposits in adventitia and inflammatory cells surrounding the media of this arteriole are seen (H&E, 200 Ҳ). Panel (b) shows the deposition of lipids and infiltration of lipid-laden foamy cells in the tunica intima and tunica media together with severe narrowing by atherosclerotic plaque, with several hemorrhagic necrotic cores (H&E, 400 Ҳ). Panel (c) indicates histologic finding with fibrous tissue, and necrotic core (H&E, 400 Ҳ). Panel (d) shows microscopic section of the coronary artery shows thickening of the vascular wall by atherosclerotic deposits and hemorrhage. (H&E, 200 Ҳ)