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. 2015 Jul 1;18(7):762–775. doi: 10.1089/jmf.2014.3341

FIG. 4.

FIG. 4.

LJ suppresses LPS-induced phosphorylation of JNK (A), ERK 1/2 (B), p38 MAPKs (C), and PI3K/Akt (D) in BV-2 microglial cells. Cells were pretreated with the indicated concentrations of LJ for 30 min and then exposed to 100 ng/mL of LPS for 1 h. The expression levels of JNK, p38, ERK 1/2 MAPK, and PI3K/Akt were evaluated by western blot analysis. Densitometric results are presented as the mean±SEM (n=3). ***P<.001 compared with the control group. #P<.05, ##P<.01, and ###P<.001 compared with the LPS-treated group. ERK 1/2, extracellular signal-regulated kinase 1/2; JNK, Jun N-terminal kinase; MAPK, mitogen-activated protein kinase; PI3K, phosphatidylinositol 3-kinases.