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A 4-week exposure to intermittent hypoxia (CIH) led to cardiomyocyte apoptosis and cardiac inflammation. (A) Heart immunohistochemistry and apoptosis assays. (B) CIH induced cardiomyocyte apoptosis. (C and D) CIH increased tumor necrosis factor-α (TNF-α) and interleukin-6 (IL-6) levels. CON, group exposed to 4 weeks of normoxia. Scale bar, 100 µm. *P<0.05 vs. normoxia group.
