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. 2015 Jun 11;56(4):700–708. doi: 10.1093/jrr/rrv026

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© The Author 2015. Published by Oxford University Press on behalf of The Japan Radiation Research Society and Japanese Society for Radiation Oncology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 6. — (A) Phospho-AKT and Phospho-ERK1/2 expression by western blot in SECs treated with or without MSC-CM for 45 min. (B) SECs were cultured in the presence of the PI3 K/Akt inhibitor (LY294002) and MEK/ERK inhibitor (U0126) with or without RT and MSC-CM, and cell apoptosis was evaluated by fluorescence-activated cell sorting (FACS) analysis. Data are shown as means ± SD. **P < 0.01 (DMEM group versus control group). ^##P < 0.01 (MSC-CM group versus DMEM group). ^&&P < 0.01 (MSC-CM group versus MSC-CM + LY294002 group). Two closed triangles: P < 0.01 (MSC-CM group versus MSC-CM + U0126 group).