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. 2015 Jul 15;26(14):2673–2684. doi: 10.1091/mbc.E15-04-0224

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© 2015 Cox et al. This article is distributed by The American Society for Cell Biology under license from the author(s). Two months after publication it is available to the public under an Attribution–Noncommercial–Share Alike 3.0 Unported Creative Commons License (http://creativecommons.org/licenses/by-nc-sa/3.0).

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FIGURE 7: — Model for endosomal control of contact inhibition through YAP:AMOT. In subconfluent cells, AMOT associates with EDTB on endosomes, allowing YAP to localize to the nucleus. When cells reach confluence, tight junction proteins cycling through endosomes associate with EDTB, allowing AMOT to sequester YAP at the endosomal membrane.