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SR-nitrite therapy improves coronary vascular reactivity by an NO-dependent mechanism in the setting of MetS and CLI. Freshly isolated coronary arteries were precontracted with PGF2α. A: dose-dependent relaxation (%relaxation) of the left anterior descending coronary artery in response to sodium nitroprusside. B: maximum relaxation at 10−5 M sodium nitroprusside. Values are means ± SE; numbers inside bars are numbers of animals per group. *P < 0.05 vs. the CLI + placebo-treated group.
