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. Author manuscript; available in PMC: 2016 Mar 1.
Published in final edited form as: Mucosal Immunol. 2015 Jan 21;8(5):1047–1059. doi: 10.1038/mi.2014.132

Figure 8.

Figure 8

C. albicans infection of the gastric mucosa is resistant to ILC dependent increases in calprotectin. Groups of rag1−/− mice were infected with C. albicans and treated with ILC-depleting antibody (anti-CD90.2) or an isotype matched control antibody (n ≥ 8). (a) Representative H&E stained stomach sections demonstrating the primary location and nature of lesions in response to infection. (b) Calprotectin subunit S100A9 levels were determined by semi-quantitative PCR on total RNA isolated from the gastric mucosa (± SEM). (c) Fungal load was determined by quantitative PCR of the ITS region of the rRNA gene of C. albicans and reported as copy number per gram of stomach tissue (± SEM). * P<0.05.