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. 2015 Jul 21;10(7):e0133190. doi: 10.1371/journal.pone.0133190

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© 2015 Kreit et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 1 — A. Phylogenetic tree of the murine Apol gene family, generated by the Gene Orthology/Paralogy prediction method available on the Ensemble server (http://www.ensembl.org). Duplication nodes are indicated by black squares. Numbers represent the duplication confidence score. B. Organization of the mouse Apol genes cluster. Underlined regions (Apol7b/9a and Apol7e/9b) share 98% of nucleotide sequence identity and likely arose from a duplication event. C. Alignment of the human ApoL6 BH3 domain with the hypothetical BH3 domain of murine ApoL proteins. Two amino acids, L and D, shown in bold letters, are functionally conserved in all the BH3 domains identified. D. Structural organization of ApoL9 proteins. The putative BH3 domain (residues 81 to 89) is shown in dark gray. The white area (residues 128 to 144) corresponds to a potential transmembrane (TM) domain.