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. 2015 Jul 24;15:542. doi: 10.1186/s12885-015-1562-9

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© Drakaki et al. 2015

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — MiR-9 signaling pathway levels in HCC tissues. a CDH1 and (b) PPARA mRNA relative expression levels in 24 HCC tumors and 11 control liver tissues assessed by real-time RT-PCR analysis. Gene expression levels were normalized to the levels of GAPDH and β-actin. Normalized gene expression levels were quantified relative to the levels of a given control tissue. c Correlation analysis between miR-9 and CDH1 mRNA levels in 24 HCC tissues. d Correlation analysis between miR-9 and PPARA mRNA levels in 24 HCC tissues. Data are represented as mean ± SE. ***P < 0.001, in comparison to control