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. 2015 Jun 30;7(7):5239–5253. doi: 10.3390/nu7075220

Figure 1.

Figure 1

AVLE concentration-dependently (100 ng/mL–100 µg/mL) induced relaxation in isolated rat aortas. (A, B) Representative traces and graph showing the relaxation induced by AVLE was markedly decreased by inhibitor of Src kinase (PP2, 20 µM); and PI3-Kinase inhibitors (wortmannin, 30 nM and LY294002, 20 µM) (C) while eNOS inhibitor (L-NAME, 100 μM,) and soluble guanylate cyclase inhibitor (ODQ, 1 μM) abolished the relaxation response by AVLE. Results are means ± SEM (n = 5–6). * p < 0.05 compared to control.