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. Author manuscript; available in PMC: 2015 Aug 3.
Published in final edited form as: Pediatr Res. 2014 Dec 17;77(3):477–483. doi: 10.1038/pr.2014.200

Table 1.

Distribution of clinical and epidemiological risk-factors for BPD in our cohort:

Variable Infants without BPD (n=375) Infants with BPD (n=284) Infants with severe BPD (n=135)

Gestational age (wk) 29 (28; 30) 26 (25; 28)* 26 (24; 27)*

Birth-weight (grams) 1190 (678; 1352) 876 (675; 1107)§ 760 (635; 991)§

Race - Caucasians 68% 77.8% 75.5%
African American 21.3% 10.2% 11.9%
Others 10.7% 12.0% 12.6%

Prenatal steroid use 85% 91.1% 91%

Male sex 48.8% 57.8%§§ 63.0%§§

Clinical chorioamnionitis 7.5% 11.7% 10.3%

Patent ductus arteriosus 22.6% 58.3%** 65.9%**

Data is represented as median ± interquartile range or as raw numbers with percentages.

*

P<0.001 (no BPD vs. BPD; severe BPD vs. others),

§

P=0.001 (no BPD vs. BPD; severe BPD vs. others),

P=0.006 (% CAU infants, no BPD vs. BPD),

§§

P<0.02 (no BPD vs. BPD; severe BPD vs. others),

**

P<0.001 (no BPD vs. BPD; severe BPD vs. others).

Clinical chorioamnionitis was diagnosed in the presence of maternal fever >38°C plus one additional criteria (uterine tenderness, malodorous vaginal discharge, maternal leukocytes >15,000 cells/mm3 or fetal heart-rate of >160/min)