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TXL inhibited inflammation and oxidative stress by inhibiting the activation of NF-κB and p47 by C16. HCMECs were grown in six-well plates for 24 h, and then were pre-incubated with TXL before stimulating with C16 for 1 h. The expression of p47, p-p47, p22, p-p22, NF-κB, and p-NF-κB was detected by Western blotting. *P < 0.05 vs. C16-untreated group; #P < 0.05 vs. C16-treated group without TXL treatment. The bars represent the mean ± SEM. from 3 independent experiments.
