Table 2. The relationship between motor performance ^a and the CAG repeats length in somatic tissues ^b .

Percentile of CAG repeats	Striatum c	Hippocampus c	Cerebellum c	Cortex c
1	P = 0.002*	P = 0.01*	P<0.0001*	P = 0.005*
5	P = 0.001*	P = 0.05*	P = 0.0006*	P = 0.05*
10	P = 0.005*	P = 0.04*	P = 0.001*	P = 0.11
20	P = 0.02*	P = 0.12	P = 0.006*	P = 0.21
30	P = 0.06	P = 0.23	P = 0.01*	P = 0.40
40	P = 0.07	P = 0.43	P = 0.03*	P = 0.54
50	P = 0.12	P = 0.42	P = 0.18	P = 0.66
60	P = 0.16	P = 0.62	P = 0.78	P = 0.74
70	P = 0.45	P = 1.00	P = 0.65	P = 0.85
80	P = 0.75	P = 0.62	P = 0.21	P = 0.85
90	P = 0.89	P = 0.08	P = 0.07	P = 1.00
95	P = 0.47	P = 0.04†	P = 0.01†	P = 0.69
99	P = 0.44	P = 0.008†	P = 0.01†	P = 0.86

* Less repeats associated with longer time on the rod

^† More repeats associated with shorter time on the rod

^a. Time on the rotarod at 20rpm

^b. All the changes in CAG length were pooled for each tissue from birth to 40 weeks, the time frame of onset.

^c. The significance of the association between motor performance and somatic expansion was determined by linear regression. The coefficients were adjusted for Hdh(Q150) genotype, ogg1 genotype, age, and gender. P = probability.