Table 2. gld-1 and puf-8 function redundantly to suppress the development of spermatogenesis-dependent tumor in hermaphrodites.

Genotype	Percent of Worms With Tumorous Germline	Total Number of Worms
Hermaphrodites grown at 20°
gld-1(q485)a	100a	194
puf-8(zh17)	0	118
gld-1(q485); puf-8(zh17)	100	134
gld-1 (RNAi); puf-8(q725)	100	48
gld-1 (RNAi); puf-8(ok302)	100	72
gld-1(RNAi); puf-8(RNAi)	100	288
rrf-1(ok589) gld-1 (RNAi); puf-8(RNAi)	100	122
gld-1(q485); puf-8(zh17); spe-6(hc49)	100	208
spe-6(hc49)	0	92
gld-1(RNAi) mek-2(q425); puf-8(RNAi)	100	174
gld-1(q485) mek-2(RNAi); puf-8(zh17)	100	44
gld-1(RNAi); puf-8(RNAi); spe-6(hc49)	100	174
Hermaphrodites grown at 25°
puf-8(zh17)	43	181
puf-8(RNAi)	42	84
rrf-1(ok589); puf-8(RNAi)	45	102
gld-1(q485/+); puf-8(zh17)	94	147
Effect of masculinization
puf-8(zh17); fem-3(q20), grown at 20°	0	324
gld-1(RNAi); puf-8(zh17); fem-3(q20), grown at 20°	100	204
gld-1(q485); fem-3(q20), grown at 20°	1	260
gld-1(q485); fem-3(q20), grown at 15°a	100a	72

^*Germline tumor in these worms was the gld-1-type. See Results and Figure 3 for more details. See Table S1 for the complete description of the genotypes, which includes the marker mutations.