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. 2015 Aug 7;13:257. doi: 10.1186/s12967-015-0622-x

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© Castañón et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — OS depending on the EGFR status stratified by the presence of LM. a A subanalysis was performed selecting those patients with liver involvement. We observed a significant difference between those patients harboring EGFR mutations (n = 9) compared to those showing wild-type EGFR (n = 46). b When selecting patients with no liver involvement, we also observed a better outcome for those harboring EGFR mutation (n = 21) compared to those with EGFR wild type NSCLC (n = 98).