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. Author manuscript; available in PMC: 2015 Aug 11.
Published in final edited form as: J Comp Neurol. 1996 Nov 11;375(2):212–224. doi: 10.1002/(SICI)1096-9861(19961111)375:2<212::AID-CNE3>3.0.CO;2-5

Fig. 1.

Fig. 1

γ-aminobutyric acid plasma membrane transporter-1 (GAT-1) immunoreactivity in the rat retina. Strong GAT-1immunostaining is in cell bodies in the proximal inner nuclear layer (INL) and in processes distributed to all regions of the inner plexiform layer (IPL). The immunoreactive pattern indicates that GAT-1 is predominantly expressed by amacrine cells. A: GAT-1 immunoreactivity in a section incubated with the GAT-1 antibody preadsorbed with 10−5 M GAT-3607–627. Immunostaining is comparable to that observed with GAT-1 antibodies alone. B: Section incubated with GAT-1 antibody. C: Lack of GAT-1 immunostaining in a control section incubated with the GAT-1 antibody preadsorbed with 10−5 M GAT-1588–599. ONL, outer nuclear layer; OPL, outer plexiform layer; GCL, ganglion cell layer. Scale bar = 25 μm.