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. 2015 Aug 14;10:143. doi: 10.1186/s13000-015-0368-z

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© Kim et al. 2015

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — EGFR and KRAS gene mutations in patients. EGFR (a) and KRAS (b) mutations were identified using the PNA clamping and direct sequencing method for 9 patients. PNA clamping analysis revealed similar results as those obtained from direct sequencing, one patient exhibited EGFR 19del on PNA clamping, but a weak mutant peak on direct sequencing