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. 2014 Sep 4;5(9):e1402. doi: 10.1038/cddis.2014.367

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Copyright © 2014 Macmillan Publishers Limited

Cell Death and Disease is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution-NonCommercial-ShareAlike 3.0 Unported License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-sa/3.0/

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The working model for the miR-193a's role in regulation of the chemoresistance of BCa by repression of HIC2, PLAU and SRSF2. (a) Expression of SRSF2, HIC2 and PLAU genes repressed by DNA methylation-regulated miR-193a-3p, is positively correlate with the chemoresistance to four indicated chemotherapeutics. (b) A network model. Lines signify the potential interactions between genes obtained from the STRING database. The green nodes indicate the upregulated genes and the red nodes indicate the downregulated genes in 5637 relative to H-bc cells. (Notch, Myc, DNA damage (DD), NF-κB and Oxidative Stress (OS)). (c) Relative expression level from RNA-seq data of the genes are summarized in the table