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. 2004 Jun 25;101(27):10132–10136. doi: 10.1073/pnas.0401900101

Fig. 5.

Fig. 5.

Amyloid oligomer reactivity in human heart failure samples. (a–c) Samples derived from normal hearts. (d) A 50-year-old Caucasian male diagnosed with dilated cardiomyopathy (DCM) 1 year before death. (e) A 33-year-old Caucasian female with HCM. Complete heart block with pacemaker placement is shown. The patient died of an arrhythmia. (f) A 57-year-old African-American female with idiopathic DCM diagnosed 6 years before death. (g) A 37-year-old Caucasian male with idiopathic DCM. The patient died of CHF. (h) A 34-year-old African-American female with nonischemic cardiomyopathy diagnosed 3 years before death from CHF. (i) A 16-year-old Caucasian male diagnosed with idiopathic DCM 2 years before death. The patient died of an arrhythmia. (j) A 45-year-old Caucasian male with family history of HCM was diagnosed as being in the dilated stage of HCM 1.5 years before death. The patient died of CHF. (k) A 22-year-old African-American male with idiopathic DCM diagnosed 6 months before death. The patient died of CHF. (l) A 32-year-old Caucasian male with muscular dystrophy identified tentatively as Becker's. He died of pneumonia with complicating episodes of cardioverted ventricular tachycardia–fibrillation.