Skip to main content
editorial
. 2015 Jun 8;6(16):13852–13853. doi: 10.18632/oncotarget.4386

Figure 1. MOD1-mediated programmed cell death through mitochondrial complex I (CI)-generated ROS.

Figure 1

Knock out of SOM3, a component of CI, and overexpression of SOM42, a negative regulator of CI, can both repress cell death and ROS accumulation phenotypes of mod1. The red color highlights the mutant mod1 and its supressors, som3 and som42.