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. Author manuscript; available in PMC: 2016 Sep 1.
Published in final edited form as: Vascul Pharmacol. 2015 Jun 18;72:9–15. doi: 10.1016/j.vph.2015.06.007

Figure 2.

Figure 2

Biological responses following stent deployment. Bare metal stents (BMS) elicit a reendothelialization response, but also induce smooth muscle cell proliferation leading to restenosis (bottom left). Drug eluting stents (DES) releasing sirolimus and derivatives inhibit smooth muscle cell proliferation, but also prevent reendothelialization. Lack of endothelial coverage results in thrombosis (bottom middle). Regeneration of the endothelial lining prevents thrombosis and inhibits smooth muscle proliferation (bottom right).