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. 2015 Aug 26;17(1):230. doi: 10.1186/s13075-015-0742-y

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© Blüml et al. 2015

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — Absence of PTEN in APCs does not affect the development of innate immunity-dependent arthritis. a, b Clinical assessment of (a) paw swelling and (b) grip strength in wt (n = 7) and myeloid pten ^-/- (n = 7) after induction of K/BxN serum transfer arthritis. Data are expressed as means ± s.d. C Quantitative histomorphometric analysis of the hind paws of wt and myeloid pten ^-/- mice 12 days after induction of K/BxN serum transfer arthritis. Data are expressed as means ± s.d. *P ≤ 0.05. d Representative histologies of hind paws of wt and myeloid pten ^-/- mice obtained 12 days after induction of K/BxN serum transfer arthritis (H&E staining, TRAP staining). Scale bars, 1 mm. APCs antigen-presenting cells, H&E hematoxylin and eosin, PTEN phosphatase and tensin homolog, TRAP tartrate-resistant acid phosphatase, wt wild-type