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. Author manuscript; available in PMC: 2016 Aug 1.
Published in final edited form as: Dev Biol. 2015 Apr 24;404(1):55–65. doi: 10.1016/j.ydbio.2015.04.015

Figure 5.

Figure 5

CAM-1 CRD is both necessary and sufficient to antagonize Wnt function in ALM polarity. Expression in a cwn-1 background of either a membrane-tethered CAM-1 CRD or full-length CAM-1 led to polarity phenotypes that were not significantly different from one another. A CAM-1 transgene that lacks the CRD failed to induce polarity defects above background. A CAM-1 transgene expressed from the muscle-specific myo-3 promoter or a CAN neuron-specific promoter induced polarity defects in a cwn-1 background. Data are presented as in Fig. 1K. N.S., not significant.