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(A) In healthy corneas, proteoglycans plays an important role of intrafibril interaction. (B) Collagenase treatment degraded protelglycan core protein, as well as collagen helix molecules in the fibril, which resulted in fibril diameter decrease. (C) Chondroitinase treatment degraded the glycosaminoglycan, which resulted in weakened intrafibril crosslinking, and damaged single fibril structure.
