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. 2015 Aug 27;6(8):e1865. doi: 10.1038/cddis.2015.239

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Copyright © 2015 Macmillan Publishers Limited

Cell Death and Disease is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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MiR-26a-GSK3β regulate sensory axon regeneration by controlling gene expression. (a) Representative images of cultured adult mouse sensory neurons expressing EGFP, EGFP+miR-26a inhibitor (anti-26a) and EGFP+anti-26a treated with the GSK3 inhibitor BIO before cell replating. All neurons were stained with anti-βIII tubulin antibody (Tuj-1, red). Scale bar=100 μm. (b) Quantification of the average length of the longest axons (normalized to the average length of the control axons, n=3). Bar graphs are shown as means±S.E.M. ***P<0.001