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. 2015 Jul 1;7(8):2117–2135. doi: 10.1093/gbe/evv129

Table 2.

Distribution of HPVs by their Clinical Presentation in Each Conglomerate Obtained by Two-Step Cluster Analysis

E6a, %
E7, %
Cluster1 Cluster2 Cluster3 Cluster1 Cluster2
MucCutAsym (n = 90) 97.8 2.2 94.4 5.6
MucL (n = 25) 100 100
GenW (n = 13) 100 100
CutW (n = 28) 3.6 96.4 3.6 96.4
E1, %
E2, %
E4, %
Cluster1 Cluster2 Cluster3 Cluster1 Cluster2 Cluster3 Cluster1 Cluster2
MucCutAsym (n = 90) 94.4 3.4 94.4 94.4 3.4 2.2 94.4 5.6
MucL (n = 25) 100 100 100
GenW (n = 13) 100 100 100
CutW (n = 28) 3.6 3.6 3.6 96.4 3.6 96.4
L2, %
L1, %
Cluster1 Cluster2 Cluster3 Cluster1 Cluster2 Cluster3
MucCutAsym (n = 90) 94.4 3.4 2.2 94.4 3.4 2.2
MucL (n = 25) 100 100
GenW (n = 13) 92.3 7.7 84.6 15.4
CutW (n = 28) 3.6 96.4 3.6 96.4

Table should be read as follows, taking E7 as an example: For E7, two-step cluster analysis identifies two different clusters: Cluster 1 spans 94.4% (n = 85) of MucCutAsym HPVs and 3.6% (n = 1) of HPVs causing CutW. Cluster 2 spans 100% (n = 25) of HPV causing mucosal lesions, 100% (n = 13) of HPVs causing GenW, 96.4% (n = 27) of HPVs causing CutW and 5.6% (n = 5) of HPVs causing MucCutAsym infections.

aCutA in E6, n = 87.