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. Author manuscript; available in PMC: 2016 Sep 10.
Published in final edited form as: Exp Cell Res. 2015 Jul 21;337(1):120–127. doi: 10.1016/j.yexcr.2015.07.017

Figure 6.

Figure 6

Myeloid-specific disruption of SOCS3 resulted in decreased pro-inflammatory mediator expressions in IgG IC-treated lungs. SOCS3fl/fl and LysM-Cre SOCS3fl/fl mice were treated intratracheally with IgG IC for 4 h, and cell-free supernatants were used to conduct ELISA to detect MIP-1α (A), and MIP-1β (B), and MCP-1 (C) expressions. Values represented means ± S. E. M. for ≥ 3 mice for each group. *, and ** suggested statistically significant difference— p < 0.05, and p < 0.01, respectively.