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Proposed model of the metabolic adaptation to the low-fat diet in the liver of Nile tilapia. In the liver of LF diet-fed Nile tilapia, accelerated glycolysis provides more substrate – acetyl-CoA for FA and TG synthesis to maintain the content of liver TG. This process is upregulated by SREBP1. The newly synthesized TG can be transported out to other peripheral tissues mediated by increased VLDL consisting of ApoB and ApoE.
