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. Author manuscript; available in PMC: 2015 Sep 10.
Published in final edited form as: Dig Dis Sci. 2014 Feb 7;59(4):886–891. doi: 10.1007/s10620-014-3038-1

Table 1.

Three clinical groups of study patients

Group 1: Hepatocellular damage (n = 229) Group 2: Obstructive/cholestatic (n = 56) Group 3: Miscellaneous (n = 38)
Chronic hepatitis C (n = 117) Primary biliary cirrhosis (n = 24) Cryptogenic (n = 29)
Alcoholic (n = 49) Primary sclerosing cholangitis (n = 29) Alpha-1-antitrypsin deficiency (n = 6)
Chronic hepatitis C plus alcoholic (n = 28) Biliary atresia (n = 2) Alagille’s syndrome(n = 1)
Chronic hepatitis B (n = 8) Sarcoidosis (n = 1) Polycystic liver disease (n = 1)
Autoimmune hepatitis (n = 12) Cystic fibrosis (n = 2) Hereditary hemorrhagic telangiectasia (n = 1)
Hemochromatosis (n = 3)
Wilson’s disease (n = 2)
Non-alcoholic steatohepatitis (n = 10)