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. 2015 Sep 10;6(1):171. doi: 10.1186/s13287-015-0154-6

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© Yamaza et al. 2015

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — SHED ameliorate the hepatic dysfunction in recipient livers of CCl₄-treated mice. a Serum assays for the hepatic function. b–f Liver fibrosis assays. b Representative images of livers. Masson Trichrome staining. Arrowheads, fibrous deposition. c Fibrotic area. d Fibrotic score. e Hydroxyproline assay in recipient livers. f Real-time RT-PCR analysis of mouse type I collagen (mCola1) mRNA in recipient livers. a, c–f n = 5 for all groups. Control, olive oil-injected group; CCl₄, CCl₄-treated group; CCl₄ + SHED, SHED-transplanted CCl₄-treated group. *P <0.05 and ***P <0.005. ns no significance. Graph bars show the mean ± SD. ALT alanine aminotransferase, ALP alkaline phosphatase, AST aspartate aminotransferase, CCl ₄ carbon tetrachloride, SHED stem cells from human exfoliated deciduous teeth