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. 2015 Sep 11;11:1371–1381. doi: 10.2147/TCRM.S88297

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© 2015 Zhao et al. This work is published by Dove Medical Press Limited, and licensed under Creative Commons Attribution – Non Commercial (unported, v3.0) License

The full terms of the License are available at http://creativecommons.org/licenses/by-nc/3.0/. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed.

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Effects of ALDH2 on Wnt/β-catenin-regulated cardiac fibrosis after MI in rats from each group.

Notes: Western blotting images of β-catenin and total and phosphorylated GSK-3β (A), Quantitative analyses of phosphorylated GSK-3β (normalized to total GSK-3β) (B), quantitative analyses of β-catenin (C), mRNA expression of Wnt-1 (D), phosphorylated GSK-3β (normalized to total GSK-3β) (E), β-catenin (F), collagen type I (G), collagen type III (H), and α-SMA (I). Data are expressed as mean ± SD, n=6 per group. *P<0.05, **P<0.01 vs the control group; ^#P<0.05, ^##P<0.01 vs the model group.

Abbreviations: ALDH2, aldehyde dehydrogenase-2; GSK-3β, glycogen synthase kinase; MI, myocardial infarction; pGSK-3β, phosphorylation of glycogen synthase kinase-3β; SD, standard deviation; α-SMA, α-smooth muscle actin.