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. Author manuscript; available in PMC: 2015 Sep 18.
Published in final edited form as: J Am Soc Hypertens. 2013 Feb 19;7(2):170–179. doi: 10.1016/j.jash.2013.01.003

Figure 2.

Figure 2

Flow diagram detailing the role of kallikrein in promoting diuresis. Kallikrein secretion, triggered by excess sodium in the connecting tubule, generates renal kinins which act on the brady-kinin receptor 2 (BK B2 R) to promote diuresis.