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. 2015 Sep 22;22(1):80. doi: 10.1186/s12929-015-0188-1

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© Lee et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — Mechanism of caffeic acid ethanolamide (CAEA) affecting cardiac oxidative stress and cardiac bioenergetics in mice subjected to two weeks subcutaneous isoproterenol (ISO) injections. a Quantification of cardiac sirtuin 1 (SIRT1) and sirtuin 3 (SIRT3) activity. b Quantification of cardiac lipid peroxidation. c Representative Western blot image of cardiac HIF-1α expression. d Densitometry of cardiac HIF-1α expression. e Quantification of cardiac lactate content. f Representative M-mode echocardiogram. g Quantification of left ventricular ejection fraction. h Quantification of cardiac ATP content. n = 6 ~ 9, **P <0.01 versus control, *P <0.05 versus control, ^# P <0.05 versus ISO, ^@ P <0.05 versus ISO + CAEA. (Nic = nicotinamide)