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. 2015 Jun 6;1(2):82–93. doi: 10.1159/000430786

Table 4.

IPA was queried with 146 genes altered on both microarray platforms in the PCP treatment group compared to the VEH control group

Ingenuity canonical pathways B-H p value Genes
Huntington's disease signaling 4.27 × 10−7 MAPK1, PIK3R1, UBE2S, SNAP25, CDK5R1, PRKCG, DNM1, BCL2L1, MTOR, PRKCI, PENK, STX16, NCOR2, NAPA, RASA1

Melatonin signaling 1.78 × 10−5 PRKACB, CAMK4, PRKCI, MAPK1, RORB, PRKCG, PRKAR1A, CAMK2G

Neuropathic pain signaling in dorsal horn neurons 1.78 × 10−5 PRKACB, CAMK4, PRKCI, MAPK1, GRIA1, PIK3R1, PRKCG, PRKAR1A, CAMK2G

Synaptic long-term potentiation 2.63 × 10−5 PRKACB, CAMK4, PRKCI, MAPK1, GRIA1, PPP3CA, PRKCG, PRKAR1A, CAMK2G

Calcium signaling 2.63 × 10−5 PRKACB, CALR, CAMK4, MAPK1, GRIA1, MEF2C, ATP2A2, PPP3CA, ATP2B2, PRKAR1A, CAMK2G

IPA canonical pathway analysis identified the following over-represented canonical pathways after correcting for multiple comparisons. B-H = Benjamini-Hochberg.