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. 2015 Sep 22;12:179. doi: 10.1186/s12974-015-0389-2

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© Jiang et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Fas^lpr mice do not develop ocular inflammation or release HMGB1 after IRBP-specific T cell transfer. Wt and Fas^lpr mice were injected with IRBP_1–20-specific T cells and examined on day 15 (a–c) post-injection. a Representative ocular histopathology with H & E staining of Wt and Fas^lpr eye sections, original magnification ×100. b Pathological score for the two groups (n = 12 mice) presented as the mean ± SEM. **p < 0.01 compared to Wt mice using the Mann-Whitney U test. c HMGB1 levels in the intraocular fluid (6 eye/group) measured by ELISA. **p < 0.01 compared to naïve mice by one-way ANOVA