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A heart‐centric MMP‐2/sPLA2 axis may modulate blood pressure homeostasis, inflammatory and metabolic gene expression, as well as the severity of fever. Future research should establish whether cardiac sPLA2 is activated secondary to preexisting inflammation or if it is the primary cause of the inflammatory state characteristic of MMP‐2 gene deficiency. MMP indicates matrix metalloproteinase; sPLA2, secreted phospholipase A2.
