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. 2015 Sep 28;8:40. doi: 10.1186/s13072-015-0032-6

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© Zhang et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Analysis of H3K4me3 and H3K27me3 at Slc17a6. a Schematic representation of the Slc17a6 gene showing the region assayed by ChIP-qPCR (−240 to −94 bp) relative to BS1 and the TSS is shown. Horizontal arrows represent PCR primers. b H3K4me3 in the male hippocampus at P21 and P87 + 120 (n = 12 mice/group, pooled). A gene desert and Gapdh2 were used as a negative control (−Ctrl) and positive control (+Ctrl), respectively. c H3K27me3 in the male hippocampus at P21 and P87 + 120 (n = 12 mice/group, pooled). A gene desert and Pax2 were used as negative and positive controls, respectively. Data are shown as mean and standard deviation. *P < 0.05 (t test, two-tailed)