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. 2015 Sep 29;12:182. doi: 10.1186/s12974-015-0401-x

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© Ji et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — The levels of PV, 4-HNE, and gp91^phox in the PFC and hippocampus. a, b Sepsis induced significant decreases in the PV level from day 3 to day 14 and significant increases in the 4-HNE level from 24 h to day 3 in the hippocampus. c–f The PV level was decreased and the gp91^phox level was increased in the hippocampus at day 14 but not at day 30 in the CLP + vehicle group compared with the sham groups, whereas apocynin treatment reversed these abnormities. There was no significant difference in the PV and gp91^phox levels in the PFC among the four groups. Data are presented as mean ± SEM (n = 6). ^# p < 0.05 vs the sham groups; *p < 0.05 vs the CLP + vehicle group