FIGURE 1.

M. tuberculosis recG functionally complements E. coli ΔrecG cells from replicative stress induced by HU. Survival of wild-type E. coli (AB1157) and E. coli ΔrecG (N3793) mutant strains were transformed with either empty vector or plasmids encoding E. coli recG and M. tuberculosis recG without (A) and with HU treatment (B). For qualitative analysis, the indicated dilutions of cells were plated and incubated overnight. The images of the plates were captured by the gel documentation system. For quantitative measurement, serially diluted cells were plated and counted after overnight incubation (C). The error bars represent standard error of the mean (S.E.) from three independent experiments.