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. 2015 Mar 6;39(4):314–321. doi: 10.1016/j.jgr.2015.02.004

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Copyright © 2015, The Korean Society of Ginseng, Published by Elsevier Ltd.

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 3 — The effects of TEA, 4-aminopyridine, apamin, and glibenclamide on pacemaker activity inhibition by GRe in cultured ICC clusters. Pretreatment with (A) TEA (10mM), (B) 4-aminopyridine (5mM), or (C) apamin (1μM) did not affect the inhibitory effects of GRe. (D) Pretreatment with glibenclamide (10μM) blocked the inhibitory effects of GRe. (E) The GRe-induced inhibitory effects were reversed by adding glibenclamide. (F) The graph summarizes the responses to GRe in the presence of potassium channel blockers. The bars represent the mean ± the standard deviation. ** p < 0.01. 4-ami., 4-aminopyridine; CTRL, control; Gliben., glibenclamide; TEA, tetraethylammonium chloride.