Figure 8.
A model for transformation by SF2/ASF. SF2/ASF (which can be blocked by specific shRNAs) activates several key targets that contribute to the transformed phenotype: induction of MNK2b promotes phosphorylation of eIF4E, which enhances cap-dependent translation; induction of the oncogenic S6K1 isoform-2 directly leads to transformation; and induction of inactive isoforms of the putative tumor suppressor BIN1 inhibits apoptosis. Other SF2/ASF targets are also likely to contribute to transformation.