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. 2015 Aug 24;182(8):714–722. doi: 10.1093/aje/kwv128

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© The Author 2015. Published by Oxford University Press on behalf of the Johns Hopkins Bloomberg School of Public Health. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

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Figure 3. — Power curves for the 4 analyses: solid curves, colorectal cancer (CRC) versus no CRC (black, unadjusted; gray, adjusted for polyps); small dashed curves, CRC versus neither CRC nor detected polyp; large dashed curves, CRC or detected polyp versus neither. Left (A, C, E): Pr(screening | family history of polyps) = 0, Pr(screening | family history of CRC) = 0, and Pr(CRC prevented | polyp detected) = 0; right (B, D, F): Pr(screening | family history of polyps) = 0.25, Pr(screening | family history of CRC) = 0.75, and Pr(CRC prevented | polyp detected) = 0.50. Top (A and B), G₁; middle (C and D), G₂; bottom (E and F), G₃. PR, probability; G_1–G₃, simulated genes related to CRC development through risk of polyps, risk of CRC but not polyps, and progression from polyps to CRC, respectively.