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. 2015 Oct 8;13:259. doi: 10.1186/s12916-015-0489-y

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© Repetto et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — s-RNY expression is induced in atherosclerotic areas and in smooth muscle foam cells. a RT-qPCR analysis of s-RNY1-5p in ApoE ^−/− aortic arches and femoral arteries. The data were normalized by U2 snRNA and are presented as mean and standard deviation (n per group = 5). b RT-qPCR analysis of the indicated s-RNYs in CRL 2797 cells (smooth muscle cells; left panel, n per group = 6), CRL 2181 cells (endothelial cells; central panel, n per group = 7), primary myocardial cells (right panel, n per group = 3) incubated for 28 h with 0.25 μM thapsigargin in combination with the indicated concentration of acetylated LDL and control. The data were normalized by U2 snRNA and are presented as mean and standard deviation. Student’s t-test: *P <0.05, **P <0.01