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. Author manuscript; available in PMC: 2016 Aug 1.
Published in final edited form as: Cytoskeleton (Hoboken). 2015 Aug;72(8):373–387. doi: 10.1002/cm.21236

Fig. 14. Loss of synectin/GIPC function does not result in ventricular fibrosis or altered VEC ultrastructural organization.

Fig. 14

(A, B) Localization of fibronectin (green) and F-actin (red) in WT (A) and synectin/GIPC −/− heart. (C, D). TEM of capillaries in WT (A) and synectin/GIPC −/− heart. Note the absence of fibrotic regions and normal VEC ultrastructure in the synectin/GIPC −/− although as shown here there are often increased numbers of surface caveolae and cytoplasmic vesicles. Bar A, B: 100 μm. Bar C, D: 500 nm