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. Author manuscript; available in PMC: 2016 Nov 12.
Published in final edited form as: Neuroscience. 2015 Sep 1;308:106–114. doi: 10.1016/j.neuroscience.2015.08.062

Figure 4.

Figure 4

Normal differentiation and maturation of oligodendrocytes in PTPRD mutant mice. Traverse spinal cords sections from P4 wild-type (A, B) and PTPRD knock-out (A′, B′) littermates were immunostained with anti-Olig2 (A, A′), anti-APC (B, B′). Similarly, spinal cords sections from P14 wild-type (C, D, E) and PTPRD knock-out (C′, D′, E′) were subjected to immunostaining with anti-Olig2 (C, C′) or anti-APC (D, D′), and in situ hybridization with PLP (E, E′). No significant difference is detected between wild-type and knock-out mutants. Scale bar: 50 μm.